In addition, it provides the signature red color of all pure krill oils. Krill oil and wild Atlantic Salmon deliver omega-3s in the mixture of triglycerides and phospholipids. Fish oil doesn't. This is the way to get omega-3s the way nature intended.
Read more about optimizing omega-3 levels with krill oil, by clicking on the button below. Marine phospholipids for skin health? We know that omega-3s are among some of the most important nutrients to support long-term health, but with all the options to choose from, how do we know which solution is best? Moreover, this article will outline the head to toe benefits of krill oil supported by science, and lastly, we will share how krill oil is both sustainable and traceable.
B2B Products Krill oil What is krill oil? Sustainability Traceability Become a customer Why choose us? About Contact. Omega-3 Phospholipids fish oil. Liked this blog post? Then we think these are just for you. October 28, The power of marine phospholipids for skin care Marine phospholipids for skin health?
July 30, What is krill oil and why should you care? In addition to long-chain n-3 PUFAs, KO also contains the antioxidant astaxanthin, which may have a possible health effect.
Furthermore, we aim to discuss the findings in relation to the possible different health effects, focusing on lipids, inflammatory markers, CVD risk, and biological function of these two sources of long-chain n-3 PUFAs. In total, 14 original papers were included in the review. Among these, seven were clinical trials and seven were animal studies. The search was limited to English literature and the search was conducted to obtain any literature published before January One of the researchers performed the literature search and both of the researchers independently extracted the data.
Seven human randomized trials — five double-blind 19 — 23 and two open-label 15 , 24 ones — investigating the effects of KO compared with FO were identified Table 1.
Three of the studies 19 , 21 , 24 included healthy subjects between 20 years and 50 years , two studies 20 , 22 included healthy overweight or obese subjects 35—64 years , and two studies 15 , 23 included healthy subjects with normal or slightly elevated lipid levels and patients with hyperlipidemia mean age: 40—50 years. All but one study only male in the study by Schuchardt et al 21 included both male and female subjects.
The change in omega-3 index after consumption of KO was two-fold higher than that with FO. A third group did not receive any supplementation. In contrast, Laidlaw et al 24 administered different amounts of oil, as well as EPA and DHA from four different n-3 supplements, in a day crossover trial with 35 healthy subjects. Among the seven studies, four studies reported the effect on plasma TGs and lipoproteins.
The response on lipoproteins did not differ between the groups in the studies by Maki et al 22 and Ulven et al. Bunea et al 23 investigated the effect of intake of KO and FO for 3 months on blood lipids in a randomized controlled study of patients with hyperlipidemia.
In contrast, subjects in the placebo group showed increased mean total cholesterol and LDL cholesterol levels. Five out of seven studies investigated the effect on other cardiovascular risk markers. However, this is not surprising as the difference in scores is calculated by the difference in plasma EPA and DHA levels. Maki et al 22 investigated the effect of KO and FO responses on glucose homeostasis, high-sensitivity C-reactive protein, F2-isoprostanes, weight, and diastolic blood pressure and demonstrated that these parameters did not differ among the groups.
The systolic blood pressure declined modestly in both the KO and MO groups, while increasing in the control group; however, only the difference between the MO and the control group was significant. Ulven et al 15 observed no statistically significant differences in the serum markers of oxidative stress and inflammation between the study groups.
Bunea et al 23 showed that both KO and FO reduced blood glucose levels, whereas placebo treatment resulted in a nonsignificant increase all within-group changes. The between-group comparison showed that intake of 1 g and 1.
Both FO and KO performed significantly better than placebo in the regulation of glucose levels. Plasma endocannabinoids have been suggested to be involved in the regulation of the homeostasis of body composition by regulating food intake and energy expenditure.
Intake of KO, but not MO or OO, significantly decreased 2-arachidonoylglycerol in obese subjects, but not in overweight subjects. There was no effect of KO, MO, or OO on arachidonoylethanolamine in either obese or overweight subjects; thus, KO seemed more efficient than FO in reducing plasma endocannabinoid levels.
Seven papers investigating the effect of KO compared with FO in animal models were identified Table 2. Tillander et al 29 used a high-fat diet model and fed the mice with similar doses of KO and FO for 6 weeks. In the study by Tou et al, 31 Sprague Dawley rats were fed a high-fat diet consisting of different marine oils, all containing different amounts of EPA and DHA, for 8 weeks.
Among the seven studies, five studies reported the effects on plasma lipids. Tillander et al 29 found no differences in plasma lipids between the FO and the KO groups after 6 weeks.
However, within the FO group, total plasma cholesterol, cholesterol ester, free cholesterol, TGs, and PLs were significantly reduced compared to the same in controls.
In contrast, Wistar rats fed the same amount of FO and KO for 1—6 weeks showed significantly decreased plasma TG and total cholesterol compared to controls, but these effects seemed to be more pronounced after KO intake compared to FO intake.
Total plasma cholesterol, free cholesterol, and HDL cholesterol were however significantly reduced in both groups compared to controls, but no differences between KO and FO were observed. In contrast, Burri et al, 28 who fed mice for 12 weeks with similar amounts of EPA and DHA, did not see any changes in plasma lipids in any of the groups. These conflicting results may be due to the longer period of supplementation and probably because the mice were lean and not fed a high fat diet, as was done by Batetta et al 27 and Vigerust et al, 26 respectively.
Vigerust et al 26 did not observe any substantial difference in levels of proinflammatory cytokines between treatment groups. Batetta et al 27 compared the effects of KO and FO on ectopic fat and inflammation in obese rats. In these obese rats, KO also seemed to have a more pronounced inhibitory effect on the endocannabinoid system compared to FO, which is in accordance with the results of the human study by Banni et al.
Ierna et al 30 used an arthritis-induced mouse model to show that clinical arthritis score and hind paw swelling were significantly reduced in the KO group compared to controls. Mice fed the KO also had lower infiltration of inflammatory cells into the joint and synovial layer hyperplasia when compared to control. Thus, in this mouse model, KO seems to be more efficient compared to FO, in the treatment of arthritis.
In four studies, the aim was to understand biological effects of KO and FO by studying gene expression levels and protein activity in the liver. They also observed that FO and KO significantly reduced the activity of enzymes catalyzing de novo lipogenesis compared to the activity in controls. Tillander et al 29 used quantitative polymerase chain reaction PCR to study changes in hepatic gene expression after KO and FO supplementation.
Thus, it seems that KO has a greater potential to promote lipid catabolism. By the use of quantitative PCR, Vigerust et al 26 showed that both KO and FO downregulated specific hepatic target genes involved in de novo lipogenesis and genes involved in cholesterol import and synthesis compared to the control-treated groups.
Long-chain n-3 PUFAs derived from KO downregulated the activity of pathways involved in hepatic glucose production as well as in lipid and cholesterol synthesis.
The data also suggested that KO increases the activity of the mitochondrial respiratory chain. FO also upregulated the cholesterol synthesis pathway, which was the opposite of the effect mediated by KO.
The problem in comparing these studies is that one study analyzed whole-blood fatty acids, while the two other studies used plasma PLs and plasma RBCs. If possible, adipose tissue biopsies should also be taken to study whether the fatty acids from KO and FO are differently incorporated into adipose tissue, as shown in the animal study by Tou et al.
The doses of KO and FO, type of study subjects, and duration of the studies showed very limited effects on lipids and inflammatory markers in human studies. Most of the studies did not see any effects between the groups. In the future, better-designed clinical studies are warranted to gain insight into the beneficial health effects of KO compared to FO. The animal studies show that there is a very small difference between KO and FO when it comes to health effects.
KO seems to be more efficient in reducing the concentration of plasma TG, liver TG, and endocannabinoids, compared to FO, in animal studies. No adverse effects were reported. Some studies have shown that krill oil may be better absorbed by the body and more effective at improving risk factors for heart disease. However, more studies are needed to confirm these findings. On the other hand, if you are willing to spend the extra money for potentially greater health benefits, you may want to consider taking krill oil.
Despite their differences, both krill oil and fish oil are great sources of DHA and EPA and have plenty of research to support their health benefits. A few simple changes to your habits can go a long way in boosting your immune health. Here are 9 tips to boost your body's natural defenses. Omega-3 fatty acids are incredibly important for your body and brain.
This article lists 17 science-based health benefits of omega-3s. Here are 13 science-based benefits of taking fish oil. It is rich in omega-3 fats that are very important for your body and brain. Health Conditions Discover Plan Connect. Share on Pinterest. What Is Krill Oil? Krill Oil Contains More Antioxidants. Krill Oil Health Benefits.
The Bottom Line. We include products we think are useful for our readers. If you buy through links on this page, we may earn a small commission. While oil from both krill and fish provide health benefits, their origin, price, and benefits may differ.
Fish oil comes from oily fish, such as tuna, herring, or sardines. Krill oil comes from a small, shrimp-like animal called krill. Krill oil has a distinctive red color, while fish oil supplements are typically yellow or gold. Krill oil is usually more expensive than fish oil. While each supplement type contains omega-3 fatty acids, taking each supplement type presents various risks and benefits. Read on to find out more.
Both krill oil and fish oil contain omega-3 fatty acids. Some of the most popular and beneficial omega-3 fatty acids are eicosapentaenoic acid EPA and docosahexaenoic acid DHA. When a person consumes these fatty acids in fish, they demonstrate supportive effects on overall heart health and a reduction in the risks of heart attack and coronary artery disease.
However, while research has shown eating whole fish can have heart-protecting benefits, scientific studies have not yet proven that taking omega-3 supplements offers the same benefits as eating fish. Drug stores and online supermarkets sell both fish oil and krill oil supplements. A study from compared the effects of fish and krill oil, finding that they resulted in similar blood levels of EPA and DHA.
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